فهرست مطالب

Shiraz Emedical Journal
Volume:4 Issue: 2, Apr 2003

  • تاریخ انتشار: 1382/05/11
  • تعداد عناوین: 8
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  • H. Sadeghi Page 1
    DKA is an important complication of the both type I and II of diabetes mellitus which is diagnosed in presence of hyperglycemia, ketonemia and acidosis. This article explains the management of DKA in patients with renal failure which include fluid therapy, insulin, potassium, bicarbonate, phosphate and treatment of underlying disease. Preventive measures should be also mentioned for the patients.
  • Z. Habibaghahi Page 2
    Multiple sclerosis (MS) and antiphospholipid antibody syndrome (APS) have many things in common. MS is probably an autoimmune disease and anticardiolipin antibody is seen in some patients especially of neuromyelitic type. Additionally bright T2-Imaging foci are occasionally detected in brain MRI of patients with APS. History of previous abortion, venous or arterial thrombosis, response to anticoagulation therapy and abnormal localization of the lesions in MRI might be helpful in the differential diagnosis. This article studies multiple sclerosis associated with anticariolipin antibody.
  • S. Izadi Page 3
    Intracranial calcification is a condition in which calcium and sometimes Iron (Ferro calcification) deposit on the wall of blood vessels at various sites of brain tissues. This occurs either within normal brain tissues or in abnormal pathologic tissues. Intracranial calcifications are usually asymptomatic but symptomatic patient’s symptoms such as abnormal movement or cerebellar symptoms can be present. Other signs and symptoms depend on underlying disorder. Intracerebral calcification may be seen on brain CT scan, magnetic resonance imaging (MRI) and occasionally plain skull films. Management of underlying disorder sometimes leads to resolution of calcifications.
  • G. A. Yosefipour *, S. A. Nabavizadeh, A. Mowla Page 4
    Bell''s palsy is a benign common neurological disorder of unknown cause. It is almost always unilateral. It is the most common disease of the facial nerve. The incidence rate is 13 34 per 100000 and it is more common in third to fifth decade with variable sex patterns. Relationship between Bell�s palsy and pregnancy, hypertension, diabetes mellitus is noted in some studies. Onset of symptoms is acute and mostly, entire paralysis occurs in 48 hours. Paralysis may be preceded by pain behind the ear for one or two days. Diagnosis of Bell�s palsy is usually made be history and physical. The most important part of the diagnosis is determinating the location of the lesion. Principles of management include eye protection during sleep, massage of the weakened muscles, medications (e.g., steroids, Acyclovir, Vitamin B complex) and surgical decompression of the facial nerve. About 80% of patients attain full recovery within a few weeks, which usually is initiated with taste recovery and followed, by motor recovery. Prognosis is probably affected by the severity of the symptoms. Concomitant diseases such as diabetes mellitus have probably no effect to the prognosis of the Bell�s palsy
  • S. M. Soleimani Page 5
  • M. H. Nikoo Page 6
  • Z. Zare *, M. H. Bagheri Page 8
    Sturge Weber Syndrome (Encephalotrigeminal Angiomatosis):This disease was described clinically in 1874 by W.Allen Struge and radiographically in 1923 by Parks Weber for the first time. The syndrome is presence of a unilateral port-wine nevus in the ophthalmic division of the trigeminal nerve, along with seizure, mental retardation, contralateral hemisensory and motor deficit. The nevus is present at birth, and involvement of the upper eyelid is a strong clue to cerebral involvement, that is accompanied by a leptomeningeal angiomatosis in the parietooccipital area on the same side, which is associated with underlying cortical gliosis and calcification.Radiographic finding consist of cortical calcification that gives rise to the characteristic tram line calcification seen on plain radiography, and also calcification, cortical atrophy and hypertrophied choroids plexus on CT scan.Treatment consist of symptomtheraphy, anticonvulsant, resection of focal cortical lesion, and finally cerebral hemishpherectomy in refractory cases. Out come is directly related to the extent of primary pathology and progress of disease and complications, so clinical follow up and monitoring for seizure, buphthalmus, glaucoma, facial deformity and neurologic deficit is essential.
  • Answer of Previous ECG Quiz
    M. H. Nikoo Page 9
    In this 12 lead ECG, you can see wondering pacemaker activity (3 different P waves, 3 different PR and HR <100).