niloufar hedayati emami

Pregnancy in the women aged less than 20 years is an important public health issue, especially in developing countries. In Iran, limited studies have focused on the effects of maternal age on gestational and neonatal outcomes to demonstrate the pregnancy outcomes in young women. The present study aimed to investigate the maternal and neonatal complications in the pregnant women aged less than 20 years. In this cross-sectional study, variables such as maternal age, neonatal birth weight, head circumference at birth, one- and five-minute Apgar scores, gestational age, low birth weight, intrauterine growth restriction (IUGR), neonatal mortality, fetal death, preterm labor and cesarean section, maternal mortality, anemia, and preeclampsia were assessed in all the subjects. Mean age of the studied women was 18.79±1.27 years, and low birth weight was reported in 17.6% of the neonates. One- and five-minute Apgar scores of

Melatonin, an important hormone secreted by the epiphysis, is a powerful anti-oxidant with a high potential to neutralize medical toxins. The goal of this study was to demonstrate the beneficial effect of melatonin on epididymal sperm and reproductive parameters in mice treated with acetylsalicylic acid (ASA).
Male adult mice were divided into four treatment groups: control, ASA, melatonin, and ASA駩媶觢. Mice were administered ASA (50 mg/kg, orally) and/or melatonin (10 mg/kg, intraperitoneally), or vehicle control, for 14 days. Sperm count, sperm motility, and sperm morphology were evaluated to assess fertility. A colorimetric assay was used to measure serum total antioxidant capacity (TAC). A sperm chromatin dispersion (SCD) test was used to assess sperm chromatin integrity. Sex hormone levels were measured by ELISA.
Compared to the control group, ASA treatment resulted in a significant decrease in sperm parameters (P Short-term administration of ASA (50 mg/kg) has adverse effects on male reproductive function in mice. Co-administration of melatonin protects against ASA-induced impairment of male reproductive function by preventing the reduction in serum TAC and testosterone levels seen with ASA treatment alone.