فهرست مطالب vafa karimnia saheb

children obesity can leads to cardiovascular disease and impairment in blood coagulation and fibrinolysis. the aim of this study was the effect of resistance training on blood coagulation and fibrinolysis factors in children. Thirty obese children (8-12 yr) voluntary participated in this study and Based on anthropometrical characteristic and body composition were divided homogenously randomly into control and training groups. The resistance training consists of ten weeks (3 times per week) with the intensity of 50-60% 1- repetition maximum. Blood samples were taken before and 48 hours after training period and were used for measurement of variables. Data was analyzed with SPSS software and ANOVA repeated measure. the results showed that levels of plasminogen activator inhibitor‐1)PAI-1(,tissue-type plasminogen activator (t-PA), Von Willebrand-factor (vWF), D-daimer, fibrinogen, interlokine-6 and body fat percentage were significantly decreased in response to the resistance training in the training group compared to the control group (P0.05). furthermore, in the post-test, the resistance training causes reduction in body fat percentage, D-dimer, vWF, fibrinogen and IL-6 levels, PAI-1 and t-PA compared to the pre-test (P the results suggested that the moderate resistance training with reducing blood coagulation factors and inflammatory factor maybe causes reduction in incidence of atherosclerosis in obese children, and can be used as proper training protocol for health maintain in obese children

The aim of this study was to evaluate the effect of an acute high intensity resistance exercise (AHIRE) on blood coagulation and fibrinolysis factors pre-and post 10 weeks resistance training in obese children. 30 obese children (8-12 yr) voluntary with body mass index 27.08±0.96 kg/m2 were divided into control and training groups. The resistance training consists of ten weeks (3 times per week) with the intensity of 40-60% 1 repetition maximum (1RM) for eight exercises. Acute high intensity resistance exercise includes 2 sets of 12 repetitions with 75%1-RM for eight exercises. Blood samples were taken 4 times before and after training period (before and immediately after acute exercise). The AHIRE causes increase in tPA and vWF and causes decrease in PAI-1. There were no signigicant changes in protein S, protein C levels, platelet count, aPPT and PT in response to AHIRE in control and training groups. D-dimer levels increased in respose to exercise in taining group only. Also, ten weeks resistnace training causes reduction in fibrinogen and IL-6 levels in response to AHIRE compared to the control group. Before resistance training the results suggested that changes in coagulation and fibrinolysis factors were similar in both control and training groups approximately, in response to AHIRE. But after adaptation to resistance training, the training group showed higher fibrinolysis factors and lower coagulation and inflammation factors in response to AHIRE, that was showed with elevation in tPA, D-dimer and vWF and reduction in IL-6 and fibrinogen levels.

The purpose of this study was to determine the effects of L-carnitine supplementation on carbohydrate (CHO) and fat metabolism after resistance exercise. In this investigation, using a double blind, randomized, crossover, repeated measure design (1 wk washout), 10 healthy resistance-trained men (Mean±SD: age, 24.03±2.00 yr; mass, 72.26±5.31 kg; height, 173.90±5.02 cm) consumed 2 g placebo or LC supplement daily for one week and then performed 6 exercises of a resistance exercise protocol. Each workout was performed for 3 sets of 12 repetitions with 55% 1RM and rest periods were kept to 60 s between all sets and workout. Expired gases were measured for calculating CHO and fat oxidation. Venous blood samples were obtained before and after supplementation, immediately after resistance exercise and after the 1st and 24th hour after resistance exercise. Samples were analyzed for markers of glucose, NEFA, glycerol and insulin. Also L-carnitine plasma concentrations were measured, before and after supplementation. The results of this study suggest that plasma carnitine concentrations increased 14% (p≤0.05) with L-carnitine supplementation, with no change in the placebo trial. Compared to the placebo trial, CHO oxidation was reduced significantly (p≤0.05) with L-carnitine supplementation immediately after resistance exercise. However there were no statistically significant differences between the L-carnitine and the placebo conditions for any of the other variables examined (p>0.05). Increased plasma insulin concentrations with L-carnitine and placebo after resistance exercise attenuated result in attenuate fat oxidation in this study.

This study examined the effects of circuit resistance exercise on fat and carbohydrate metabolism during endurance exercise in overweight men.

Ten overweight men (mean±SD: age, 28.8±4.8 yr; BMI, 28.2±1.4 kg/m2; vo2max, 23.3±3.7 ml/kg/min) performed two types of exercise regimens: 1) Endurance exercise (E), and 2) Circuit resistance exercise, followed by endurance exercise (RE). Circuit resistance exercise consisted of six stations, each with three circuits at 50% of 1-RM (one repetition maximum), and a total exercise time of 21 min. Endurance exercise consisted cycle ergometer exercise at 60% of the maximal oxygen uptake for 30 min. Expired gases at rest, before and during endurance exercise were measured for calculating fat and carbohydrate oxidation. Venous blood samples were taken at rest, before and immediately after endurance exercise. Blood samples were analyzed for glucose, nonestrified fatty acids (NEFA), malonyl COA (MCOA), glycerol and insulin.

Plasma glycerol concentrations during endurance exercise increased 53% in RE vs. 46% in the E group (P<0.001). Concentration of plasma glucose before endurance exercise was higher in RE than in the E group (P<0.001). In RE exercise, although fat oxidation through the 30-min endurance exercise (mean value) was greater than in the E regimen, there was no significant difference between the two groups (P>0.05). Also, no significant difference was observed in the NEFA, MCOA, insulin, carbohydrate oxidation responses (P>0.05).

Lipolysis during the submaximal exercise is enhanced by prior circuit resistance exercise in overweight men.