جستجوی مقالات مرتبط با کلیدواژه "anthropometric measurement" در نشریات گروه "پزشکی"

Neck circumference (NC) measurement has been recently developed as a simple and time-saving anthropometric method for predicting cardiovascular risk. Asymptomatic hyperuricemia is known as one of the cardiovascular risk factors. This study investigated the association between NC and asymptomatic hyperuricemia among the general population of Korea.

This cross-sectional study examined data from 7,629 participants in the 2019–2020 Korea National Health and Nutrition Examination Survey. Multivariable logistic regression analysis was used to identify the factors associated with hyperuricemia.

Approximately 10% of participants corresponded to hyperuricemia group who were likely to be male, have more comorbidities, poorer habits, and larger NC. Multivariable regression analysis revealed that NC was significantly associated with hyperuricemia in women (OR 1.17; 95% CI 1.06-1.30), but not in men (OR 1.04; 95% CI 0.96-1.12).

A large NC is independently correlated with hyperuricemia among Korean women.

It is well known that dyslipidemia is related to cardiovascular disease, dietary aspect and obesity play an important role in CVD risk factor. In the present study, we investigated the relationship between calorie intake, anthropometrical parameters and serum lipids in apparently healthy women.
The subject were 90 healthy women aged between 20-55 who were taken part in diabetes screening plan of Shiraz University of Medical Sciences in 2011 anthropometric measurement were done by standard methods. Lipid profile was measured after 10 to 12 hours overnight fasting. Dietary calorie intake was assessed using 24h food recall. The bivariate associations between variables were examined with the Pearson correlation analysis and comparison of the calorie groups was done by t-test. Significance was accepted at P No significant relationships were seen between calorie intake, anthropometric measurements and serum lipids. Although TG level was positively correlated with BMI (r=0.4, P=0.000), WC (r=0.408, P=0.00) and WHR (r=0.33, P=0.003).
Higher calorie intake was not significantly correlated with cardiovascular risk factor including Higher BMI,WC,WHR and abnormal serum lipids, but higher BMI,WC,WHR were directly associated with TG in healthy people. Although the association between these parameters and TG may be explained by insulin resistance, the lack of a significant association between anthropometric measurements and LDL in healthy people and limited studies in relation to net calorie and these variables remains an unexpected finding requiring further investigation.

It is well recognized that an increased body weight is often associated with increased blood pressure. Moreover, leptin an adipocyte-derivedhormone is strongly suggested to have an important role in pathogenesis of the obesity. We aimed to evaluate the levels of serum leptin in association with obesity and hypertension in a sample of Iranian obese children.
Children from all the primary schools of a distinct of Tehran were screened for obesity. Children with a waist circumference equal to or above 90th percentile for their age and height were invited for the study. Anthropometric measurements were done and blood samples for fasting serum leptin levels were collected from 563 enrolled obese children. Multivariate linear regression analysis was used to evaluate relationship of various factors with obesity.
Mean Serum leptin levels were 8.65 ±2.18 (ng/ml). Serum Leptin levels were higher in girls than boys (P=0.009). There were significant correlations between body mass index (BMI) and serum Leptin levels, child age, systolic and diastolic blood pressure. Systolic blood pressure and diastolic blood pressure lost their association with serum Leptin level in multivariate linear regression analysis.
BMI is independently associated with Leptin levels among obese children. This may affirm a role for this hormone in the pathogenesis of childhood obesity. It seems unlikely that plasma Leptin be a major mediator of association between obesity and hypertension. However, severe hyperleptinemia may act as a risk factor for increased blood pressure.

Leptin is a 16-kD protein which is secreted from white adipocytes and, its discovery has generated enormous interest in the regulation of energy balance. Leptin has beenimplicated in the regulation of food intake, energy expenditure, andwhole-body energy balance in animals and human. Plasma leptin levels correlate with fat storages and respond to changes in energy balance. It was initially proposed that leptin serves a primary role as an anti-obesity hormone, but this role is commonly thwarted by leptin resistance. The profoundeffects of leptin on regulating body energy balance, make it as a primecandidate for drug therapies of obesity in humans and animals. Despite the recent achievements in unearthing the role of leptin in the pathophysiology of obesity, many important questions still remained that must be responded. More studies with follow-up designs and genetic evaluations are warranted to understand the comprehensive role of leptin in human. In this letter we have a review of known effects of leptin on human obesity up to now.