جستجوی مقالات مرتبط با کلیدواژه « palsy » در نشریات گروه « پزشکی »

To evaluate the visual evoked potentials in patients with spastic cerebral palsy. Patients and

Fifty children with spastic cerebral palsy were selected. They were all male in age range of l0 to 13 years. The visual evoked potential using the checker board stimulation method was used to evaluate the visual pathway of patients. Latency (msec) and amplitude (μv) of VEP, P100 peak was measured for all participants. The same procedure was repeated for fifty age and sex matched healthy individuals as the control group. The results obtained in two groups were compared to look for probable differences.

There was no statistically significant difference between the case and control groups regarding their mean age. The mean latencies for VEP, P100 peak in the case and control groups were 115 ± 15 msec and 95 ± 5 msec respectively (P < 0.001). The mean amplitudes for VEP, P100 peak were 82 ± 0.7 μv and 5 ± 2.15 μv in the case and control groups respectively (P < 0.001) .

Visual evoked potential is a suitable technique to check the visual pathway of patients with spastic cerebral palsy. The pathway shows pathological changes in patients with spastic cerebral palsy, which can be monitored using visual evoked potential recording.

Acute facial nerve palsy secondary to neuroendocrine adenoma of the middle ear (NAME) is a rare disorder. There is only one case report in the literature describing similar findings.   Case Report: A 50-year-old man initially presented to ENT clinic with a right-sided middle ear mass and normal facial nerve function. Over the next six days, he developed House-Brackmann grade II facial paralysis. He underwent urgent surgical exploration of the tympanic cavity and excision of the middle ear mass via a post-auricular approach. Histopathological and immunohistochemical analysis revealed NAME. Three weeks after the surgery, facial nerve function returned to normal. No recurrence was found at a 3-year follow-up.   Acute onset facial palsy induced by NAME is an extremely rare disorder. For a patient already affected by hearing impairment resulted from middle ear mass, facial weakness can have a significant additional detrimental impact on their wellbeing. The early complete excision of tumor is recommended not only as a curative treatment but also restoration of facial function.

Posterior interosseous nerve (PIN) palsy gives rise to various symptoms. The PIN due to lipoma is rare. Here, we report a case of progressive loss of finger extension due to a giant painless parosteal lipoma.

A 52-year-old patient with progressive weakness of finger extension with diagnosis of posterior interosseous nerve (PIN) palsy was referred to our clinic. Electrophysiological studies showed a right PIN neuropathy at the level of the forearm. The patient had no history of trauma of the hand. The patient underwent surgery for tumor removal and nerve decompression. The loss of finger extension due to PIN palsy gradually recovered after tumor removal and the nerve recovered from the motor deficits. The patient showed no recurrence of motor deficit after the surgery in the follow-up.

Since PIN palsy may arise from lipoma early surgical exploration and the excision of deep-seated lipoma in the proximal forearm is recommended in order to avoid permanent damage to PIN or other branches of the radial nerve.

Mobius syndrome is a rare congenital disorder with the preliminary diagnostic criteria of congenital facial and abducent nerve palsy. Involvement of other cranial nerves, too, is common. Prevalence rate of this syndrome is approximately 1 in 100,000 neonates. It is of unknown etiology with sporadic occurrence. However, data regarding the occurrence rate in India is limited. Features such as orofacial malformations, limb defects, and musculoskeletal, behavioral, and cognitive abnormalities might be associated. A thorough evaluation to identify the condition and establishing an adequate treatment plan is of utmost important in this condition. We are reporting clinical and radiographic features of Mobius syndrome in two cases along with unusual findings of limb and neck deformity.

To evaluate the effect of botulinum toxin-A as an alternate to surgery in acute complete sixth nerve palsy and to shorten the recovery period.

Thirty patients with acute complete sixth nerve palsy received 1-10 units of botulinum toxin-A (Dysport) injection in the medial rectus muscle within one month from the onset of palsy. Toxin was injected directly into the muscle belly under local (25 cases) or general (5 cases) anesthesia. At the 1st, 7th, 30th, 90th, and 180th day followup, binocular field of vision, abduction and any residual deviation were measured.

Patients aged between 9mo to 70yrs. 24 (80%) patients had significant improvement in abduction after 3 months and 6 (20%) had <10° abduction. Among treatment failures, 2 were traumatic and 2 were tumoral. Binocular diplopia free field was >75° in 22 (73%). 22 (73%) had no residual esotropia and other 8 patients (27%) had 10-50° residual esotropia which required surgery. No cases of exotropia or globe perforation were encountered.

Injection of botulinum toxin-A is a simple and safe way of treating acute complete sixth nerve palsy eliminating the need for invasive surgical manipulation in majority of cases. It can eliminate diplopia during acute stage of palsy in the cases of spontaneous recovery.