Effects of Ginger Inflammatory Supplement on the Cellular Stress Proteins Response Following the Progressive Strength Training in Volleyball Men

Various researchers indicate that strength training induces inflammation, stress and cellular damage. However, effects of the progressive strength training program and oral ginger supplement on stress protein are not well known. The aim of this study was investigate effect of short-term ginger supplement on heat shock protein70kda (HSP72) and total protein (TP) following progressive strength training in volleyball trained men. Twenty subjects in a longitudinal design were divided randomly into two groups; the progressive strength training group (Experimental group) with and without ginger supplementation, and or the Placebo group. The experimental group orally received 3 capsules (each capsule contained 1000 mg) of ginger rhizome powder. Progressive strength training started their 1st set of 10 repetitions at 50% of 10 RM, the 2nd set of 10 at 75% of 10 RM, and the 3rd set of 10 at 10 RM. The placebo group performed their sets in similar method. Blood samples were collected at before, immediate and 24hr after progressive strength training.ANOVA in repeated measure showed progressive strength training in both the experimental and placebo groups resulted in a significant increase of the HSP72 and TP in immediate (P≤0.001, both) and 24hr after progressive strength training, as compared to baseline (resting) levels (P≤0.001, 0.019, respectively). Ginger supplementation, although cause attenuates the increasing levels in the aforesaid markers, there was no significant difference in the resting levels of the markers, as compared to before the ginger supplementation (P≤0.634, 0.862, respectively). These results demonstrate 7 consecutive days of the ginger supplementation in doses of 3 gr per day was not beneficial in inhibition cellular stress induced by the progressive strength training. However, further studies with longer periods of the ginger supplementation should be performed in order to identify the significant effect of nondrug intervention on stress proteins.