هستی شری زاده

Due to the high prevalence of obesity and fatty liver disease caused by obesity, it is necessary to find an effective and useful solution. Therefore, the aim of this study was to examine the effect of 12 weeks of TRX training on fatty liver enzymes in obese and overweight female.

Totally, 30 overweight and obese female (BMI=33.24±4.7) who had no history of exercise or certain diseases were selected as a purposive sampling. Then, they were randomly divided into two groups of TRX training (n=15) and control (n=15). The training protocol included resistance training in the form of TRX suspension training, which was performed three days a week on non-consecutive days for 12 weeks, and each session lasted 60 minutes. Body weight, height and levels of liver function tests (LFTs) along with hepatic steatosis index (HSI) were measured 48 hours before and after the training period.

It was found that body weight (p=0.001) and BMI (p=0.001) in the exercise group decreased significantly compared to the control group. However, there was no statistically significant difference between the two groups in the serum levels of LFTs as well as HSI.

It seems that although TRX resistance training for 12 weeks has a beneficial effect on improving body weight and BMI, this training intervention has no effect on LFTs and HSI in overweight and obese women.

Skeletal muscle is a heterogeneous tissue composed of fast and slow fibers. One of the features of skeletal muscle is the highly organized structure that provides variety of functional capabilities ofthis tissue. Muscle fibers are also able to regulate their phenotypic properties in response to different functional needs. Types of muscle fibers can be identified by differences in their structural and functional properties. The main difference between muscle fibers is related to the contractile fibers of actin and myosin. Actin is involved in important cellular processes, including muscle contraction. Myosin is a molecular engine that converts the chemical energy released by ATP into mechanical energy. Energy production in myosin occurs through a number of ATPase in the myosin heavy chain. The myosin heavy chain isoform determines the maximum velocity and shortening force with respect to the cross-sectional area of each muscle fiber. Differences in energy costs and ATP phosphorylation potentials in different types of fast and slow fibers can be effective in the exchange of MHC isoforms. It seems that exercise can change the MHC isoforms through various mechanisms and consequently change the muscle fiber phenotype. In this review, we persent the potential effect of exercise on MHC isoforms.